Number of items: 11.
    Jewett, E., Arnott, G., Connolly, L., Vasudevan, N.  ORCID: https://orcid.org/0000-0003-4326-3938 and Kevei, E.
ORCID: https://orcid.org/0000-0003-4326-3938 and Kevei, E.  ORCID: https://orcid.org/0000-0002-0560-9208
  
(2022)
Microplastics and their impact on reproduction—can we learn from the C. elegans model?
    Frontiers in Toxicology, 4.
    
        
      
     ISSN 2673-3080
  
doi: 10.3389/ftox.2022.748912
ORCID: https://orcid.org/0000-0002-0560-9208
  
(2022)
Microplastics and their impact on reproduction—can we learn from the C. elegans model?
    Frontiers in Toxicology, 4.
    
        
      
     ISSN 2673-3080
  
doi: 10.3389/ftox.2022.748912
  
    Chandler, R. J., Cogo, S., Lewis, P. A. and Kevei, E.  ORCID: https://orcid.org/0000-0002-0560-9208
  
(2021)
Modelling the functional genomics of Parkinson’s in Caenorhabditis elegans: LRRK2 and beyond.
    Bioscience Reports, 41 (9).
    
         BSR20203672.
      
     ISSN 0144-8463
  
doi: 10.1042/BSR20203672
ORCID: https://orcid.org/0000-0002-0560-9208
  
(2021)
Modelling the functional genomics of Parkinson’s in Caenorhabditis elegans: LRRK2 and beyond.
    Bioscience Reports, 41 (9).
    
         BSR20203672.
      
     ISSN 0144-8463
  
doi: 10.1042/BSR20203672
  
    Herzog, L. K., Kevei, É.  ORCID: https://orcid.org/0000-0002-0560-9208, Marchante, R., Böttcher, C., Bindesbøll, C., Lystad, A. H., Pfeiffer, A., Gierisch, M. E., Salomons, F. A., Simonsen, A., Hoppe, T. and Dantuma, N. P.
  
(2020)
The Machado–Joseph disease deubiquitylase ataxin‐3 interacts
with LC3C/GABARAP and promotes autophagy.
    Aging Cell, 19 (1).
    
         e13051.
      
     ISSN 1474-9718
  
doi: 10.1111/acel.13051
ORCID: https://orcid.org/0000-0002-0560-9208, Marchante, R., Böttcher, C., Bindesbøll, C., Lystad, A. H., Pfeiffer, A., Gierisch, M. E., Salomons, F. A., Simonsen, A., Hoppe, T. and Dantuma, N. P.
  
(2020)
The Machado–Joseph disease deubiquitylase ataxin‐3 interacts
with LC3C/GABARAP and promotes autophagy.
    Aging Cell, 19 (1).
    
         e13051.
      
     ISSN 1474-9718
  
doi: 10.1111/acel.13051
  
    Kevei, E.  ORCID: https://orcid.org/0000-0002-0560-9208, Pokrzywa, W. and Hoppe, T.
  
(2017)
Repair or destruction: an intimate liaison between ubiquitin ligases and molecular chaperones in proteostasis.
    FEBS Letters, 591 (17).
    
         pp. 2616-2635.
      
     ISSN 0014-5793
  
doi: 10.1002/1873-3468.12750
ORCID: https://orcid.org/0000-0002-0560-9208, Pokrzywa, W. and Hoppe, T.
  
(2017)
Repair or destruction: an intimate liaison between ubiquitin ligases and molecular chaperones in proteostasis.
    FEBS Letters, 591 (17).
    
         pp. 2616-2635.
      
     ISSN 0014-5793
  
doi: 10.1002/1873-3468.12750
  
    Tawo, R., Pokrzywa, W., Kevei, É.  ORCID: https://orcid.org/0000-0002-0560-9208, Akyuz, M. E., Balaji, V., Adrian, S., Höhfeld, J. and Hoppe, T.
  
(2017)
The ubiquitin ligase CHIP integrates proteostasis and aging by regulation of insulin receptor turnover.
    Cell, 169 (3).
    
         470-482.e13.
      
     ISSN 1097-4172
  
doi: 10.1016/j.cell.2017.04.003
ORCID: https://orcid.org/0000-0002-0560-9208, Akyuz, M. E., Balaji, V., Adrian, S., Höhfeld, J. and Hoppe, T.
  
(2017)
The ubiquitin ligase CHIP integrates proteostasis and aging by regulation of insulin receptor turnover.
    Cell, 169 (3).
    
         470-482.e13.
      
     ISSN 1097-4172
  
doi: 10.1016/j.cell.2017.04.003
  
    Ackermann, L., Schell, M., Pokrzywa, W., Kevei, E.  ORCID: https://orcid.org/0000-0002-0560-9208, Gartner, A., Schumacher, B. and Hoppe, T.
  
(2016)
E4 ligase–specific ubiquitination hubs coordinate DNA double-strand-break repair and apoptosis.
    Nature structural & molecular biology, 23 (11).
    
         pp. 995-1002.
      
     ISSN 1545-9985
  
doi: 10.1038/nsmb.3296
ORCID: https://orcid.org/0000-0002-0560-9208, Gartner, A., Schumacher, B. and Hoppe, T.
  
(2016)
E4 ligase–specific ubiquitination hubs coordinate DNA double-strand-break repair and apoptosis.
    Nature structural & molecular biology, 23 (11).
    
         pp. 995-1002.
      
     ISSN 1545-9985
  
doi: 10.1038/nsmb.3296
  
    Franz, A., Kevei, É.  ORCID: https://orcid.org/0000-0002-0560-9208 and Hoppe, T.
  
(2015)
Double-edged alliance: mitochondrial surveillance by the UPS and autophagy.
    Current Opinion in Cell Biology, 37.
    
         pp. 18-27.
      
     ISSN 0955-0674
  
doi: 10.1016/j.ceb.2015.08.004
ORCID: https://orcid.org/0000-0002-0560-9208 and Hoppe, T.
  
(2015)
Double-edged alliance: mitochondrial surveillance by the UPS and autophagy.
    Current Opinion in Cell Biology, 37.
    
         pp. 18-27.
      
     ISSN 0955-0674
  
doi: 10.1016/j.ceb.2015.08.004
  
    Livnat-Levanon, N., Kevei, É.  ORCID: https://orcid.org/0000-0002-0560-9208, Kleifeld, O., Krutauz, D., Segref, A., Rinaldi, T., Erpapazoglou, Z., Cohen, M., Reis, N., Hoppe, T. and Glickman, M. H.
  
(2014)
Reversible 26S proteasome disassembly upon mitochondrial stress.
    Cell reports, 7 (5).
    
         pp. 1371-1380.
      
     ISSN 2211-1247
  
doi: 10.1016/j.celrep.2014.04.030
ORCID: https://orcid.org/0000-0002-0560-9208, Kleifeld, O., Krutauz, D., Segref, A., Rinaldi, T., Erpapazoglou, Z., Cohen, M., Reis, N., Hoppe, T. and Glickman, M. H.
  
(2014)
Reversible 26S proteasome disassembly upon mitochondrial stress.
    Cell reports, 7 (5).
    
         pp. 1371-1380.
      
     ISSN 2211-1247
  
doi: 10.1016/j.celrep.2014.04.030
  
    Kevei, E.  ORCID: https://orcid.org/0000-0002-0560-9208 and Hoppe, T.
  
(2014)
Ubiquitin sets the timer: impacts on aging and longevity.
    Nature structural & molecular biology, 21 (4).
    
         pp. 290-292.
      
     ISSN 1545-9985
  
doi: 10.1038/nsmb.2806
ORCID: https://orcid.org/0000-0002-0560-9208 and Hoppe, T.
  
(2014)
Ubiquitin sets the timer: impacts on aging and longevity.
    Nature structural & molecular biology, 21 (4).
    
         pp. 290-292.
      
     ISSN 1545-9985
  
doi: 10.1038/nsmb.2806
  
    Segref, A., Kevei, É.  ORCID: https://orcid.org/0000-0002-0560-9208, Pokrzywa, W., Schmeisser, K., Mansfeld, J., Livnat-Levanon, N., Ensenauer, R., Glickman, M. H., Ristow, M. and Hoppe, T.
  
(2014)
Pathogenesis of human mitochondrial diseases is modulated by reduced activity of the ubiquitin/proteasome system.
    Cell Metabolism, 19 (4).
    
         pp. 642-652.
      
     ISSN 1550-4131
  
doi: 10.1016/j.cmet.2014.01.016
ORCID: https://orcid.org/0000-0002-0560-9208, Pokrzywa, W., Schmeisser, K., Mansfeld, J., Livnat-Levanon, N., Ensenauer, R., Glickman, M. H., Ristow, M. and Hoppe, T.
  
(2014)
Pathogenesis of human mitochondrial diseases is modulated by reduced activity of the ubiquitin/proteasome system.
    Cell Metabolism, 19 (4).
    
         pp. 642-652.
      
     ISSN 1550-4131
  
doi: 10.1016/j.cmet.2014.01.016
  
    Varga, J., Tóth, B., Kevei, É.  ORCID: https://orcid.org/0000-0002-0560-9208, Palágyi, A. and Kozakiewicz, Z.
  
(2000)
Analysis of genetic variability within the genus Petromyces.
    Antonie van Leeuwenhoek, 77 (1).
    
         pp. 83-89.
      
     ISSN 1572-9699
  
doi: 10.1023/A:1002475816014
ORCID: https://orcid.org/0000-0002-0560-9208, Palágyi, A. and Kozakiewicz, Z.
  
(2000)
Analysis of genetic variability within the genus Petromyces.
    Antonie van Leeuwenhoek, 77 (1).
    
         pp. 83-89.
      
     ISSN 1572-9699
  
doi: 10.1023/A:1002475816014
  
This list was generated on Thu Oct 30 23:39:49 2025 UTC.